B
Brand Nizzle
Guest
Alright, this was posted in another thread, but it needs to be brought up again. I am considering completely ABOLISHING soy from my diet because of this post. Somebody, anybody, please tell me this is bullshit, pahlease. = (
Soy is poison, pure and simple!
For those of you here who claim that there are pro's and con's to the Soy debate in an effort to somehow validate the acceptance of Soy, the fact is that this methodology is intellectually dishonest and denies multiple verifiable facts.
FACT:
Soy protein has a degree of muscle building value in terms of being a highly bioavailable protein, etc, etc.
FACT:
Despite this fact, there is an abundance of research that consistently supports the multiple negative aspects that far outweigh the bioavailability of Soy.
The following have been CONSISTENTLY observed by researchers in Australia, Japan and the US:
* Decrease in total testosterone.
* Lowering of total sperm count.
* A direct causal decrease in male babies testicular growth and hormone development.
* Causes an observable decrease of intestinal development in babies.
* Has estrogen mimicking (genestein and daidzen) compounds in it. (If you'd like to debate this, bring it!)
The following empirical evidence speaks for itself. The largest male consumers of soy milk are those in South East Asia. There are obvious exceptions, however the vast majority of males living in South East Asia, are physically smaller, with less muscular builds than their European counterparts. Not surprisingly, they also exhibit less facial and body hair growth. As such, the implication is obvious!
1. Atanassova N (2000). Comparative Effects of Neonatal Exposure of Male Rats to Potent and Weak (Environmental) Estrogens on Spermatogenesis at Puberty and the Relationship to Adult Testis Size and Fertility: Evidence for Stimulatory Effects of Low Estrogen Levels. Endocrinology Vol. 141, No. 10 3898-3907
2. Chorazy PA (1995). Persistent hypothyroidism in an infant receiving a soy formula: case report and review of the literature. Pediatrics Jul: 96 (1 Pt 1): 148-50
3. Irvine CHG (1998). Phytoestrogens in soy-based infant foods: concentrations, daily intake, and possible biological effects. Proc Soc Exp Biol Med1998 Mar; 217 (3): 247-53)
4. Lohrke B (2001). Activation of skeletal muscle protein breakdown following consumption of soybean protein in pigs. Br J Nutr 2001 Apr; 85 (4): 447-57
5. Nagata C (2000). Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men. Nutr Cancer; 36 (1): 14-8
6. Newbold RR (2001). Uterine Adenocarcinoma in Mice Treated Neonatally with Genistein. Cancer Research 61, 4325-4328
7. Pollard M (2000). Prevention of spontaneous prostate-related cancer in Lobund-Wistar rats by soy protein isolate/isoflavone diet. Prostate 2000 Oct 1; 45 (2): 101-5
8. Strauss L (1998). Genistein exerts estrogen-like effects in male mouse reproductive tract. Mol Cell Endocrinol Sep 25; 144 (1-2): 83-93
9. Weber KS (2001). Dietary soy-phytoestrogens decrease testosterone levels and prostate weight without altering LH, prostate 5alpha-reductase or testicular steroidogenic acute regulatory peptide levels in adult male Sprague-Dawley rats. J Endocrinol Sep; 170 (3): 591
Soy is poison, pure and simple!
For those of you here who claim that there are pro's and con's to the Soy debate in an effort to somehow validate the acceptance of Soy, the fact is that this methodology is intellectually dishonest and denies multiple verifiable facts.
FACT:
Soy protein has a degree of muscle building value in terms of being a highly bioavailable protein, etc, etc.
FACT:
Despite this fact, there is an abundance of research that consistently supports the multiple negative aspects that far outweigh the bioavailability of Soy.
The following have been CONSISTENTLY observed by researchers in Australia, Japan and the US:
* Decrease in total testosterone.
* Lowering of total sperm count.
* A direct causal decrease in male babies testicular growth and hormone development.
* Causes an observable decrease of intestinal development in babies.
* Has estrogen mimicking (genestein and daidzen) compounds in it. (If you'd like to debate this, bring it!)
The following empirical evidence speaks for itself. The largest male consumers of soy milk are those in South East Asia. There are obvious exceptions, however the vast majority of males living in South East Asia, are physically smaller, with less muscular builds than their European counterparts. Not surprisingly, they also exhibit less facial and body hair growth. As such, the implication is obvious!
1. Atanassova N (2000). Comparative Effects of Neonatal Exposure of Male Rats to Potent and Weak (Environmental) Estrogens on Spermatogenesis at Puberty and the Relationship to Adult Testis Size and Fertility: Evidence for Stimulatory Effects of Low Estrogen Levels. Endocrinology Vol. 141, No. 10 3898-3907
2. Chorazy PA (1995). Persistent hypothyroidism in an infant receiving a soy formula: case report and review of the literature. Pediatrics Jul: 96 (1 Pt 1): 148-50
3. Irvine CHG (1998). Phytoestrogens in soy-based infant foods: concentrations, daily intake, and possible biological effects. Proc Soc Exp Biol Med1998 Mar; 217 (3): 247-53)
4. Lohrke B (2001). Activation of skeletal muscle protein breakdown following consumption of soybean protein in pigs. Br J Nutr 2001 Apr; 85 (4): 447-57
5. Nagata C (2000). Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men. Nutr Cancer; 36 (1): 14-8
6. Newbold RR (2001). Uterine Adenocarcinoma in Mice Treated Neonatally with Genistein. Cancer Research 61, 4325-4328
7. Pollard M (2000). Prevention of spontaneous prostate-related cancer in Lobund-Wistar rats by soy protein isolate/isoflavone diet. Prostate 2000 Oct 1; 45 (2): 101-5
8. Strauss L (1998). Genistein exerts estrogen-like effects in male mouse reproductive tract. Mol Cell Endocrinol Sep 25; 144 (1-2): 83-93
9. Weber KS (2001). Dietary soy-phytoestrogens decrease testosterone levels and prostate weight without altering LH, prostate 5alpha-reductase or testicular steroidogenic acute regulatory peptide levels in adult male Sprague-Dawley rats. J Endocrinol Sep; 170 (3): 591