sugar + coconut oil = bad? (study)

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saw this on cnn

"Just one meal high in saturated fat may damage blood vessels and hinder the ability of HDL cholesterol to protect arteries. Normally, HDL guards blood vessels from inflammation that contributes to artery-clogging plaque, says Stephen Nicholls, M.D., a cardiologist at the Cleveland Clinic Foundation.

Not so after a meal high in saturated fat. When Nicholls and colleagues fed 14 healthy volunteers two meals of carrot cake and a milk shake-one made with highly saturated coconut oil and one with polyunsaturated safflower oil-two things happened: The ability of blood vessels to expand and contract (a sign of healthy arteries) and the anti-inflammatory action of HDL were impaired for as much as six hours after the high saturated fat meal. In contrast, when the cake and milk shake were made with polyunsaturated fat, arterial and HDL functions improved. Just how much saturated fat was in that test meal?

"We likened it to people eating a double cheeseburger, fries, and a shake, which, unfortunately, is not that uncommon a meal," Nicholls says. CookingLight.com: Remodel your meals"

Fast facts on fats: What to eat, what to avoid - CNN.com

Whats you guys' take?
 
This is the study by the way:

Consumption of saturated fat impairs the anti-inflammatory properties of high-density lipoproteins and endothelial function.

Nicholls SJ, Lundman P, Harmer JA, Cutri B, Griffiths KA, Rye KA, Barter PJ, Celermajer DS.

OBJECTIVES: The purpose of this study was to investigate the influence of dietary fatty acids on the anti-inflammatory properties of high-density lipoproteins (HDL) and vascular function. BACKGROUND: The effect of dietary fatty acids on atherogenesis remains uncertain. METHODS: Fourteen adults consumed an isocaloric meal containing either a polyunsaturated or a saturated fat on 2 occasions. The effects of post-prandial HDL on endothelial cell expression of intercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion molecule-1 (VCAM-1) were determined. Flow-mediated dilation (FMD) and microvascular reactivity were assessed before and 3 and 6 h after the meal. RESULTS: Plasma triglycerides, insulin, and nonesterified fatty acids rose after the meals. The HDL collected 6 h after the saturated meal were less effective than HDL isolated from fasting plasma in terms of their ability to inhibit expression of ICAM-1 and VCAM-1, whereas HDL collected 6 h after the polyunsaturated meal had an inhibitory activity that was greater than that of HDL collected from fasting plasma (p < 0.004 and p = 0.01 for comparison of effect of meals on ICAM-1 and VCAM-1, respectively). Post-hyperemic microvascular flow significantly increased at 3 h after the polyunsaturated meal by 45 +/- 14% and by 21 +/- 11% after the saturated meal. The FMD decreased 3 h after the saturated meal by 2.2 +/- 0.9% (p< 0.05 compared with baseline) and by 0.9 +/- 1% after the polyunsaturated meal. CONCLUSIONS: Consumption of a saturated fat reduces the anti-inflammatory potential of HDL and impairs arterial endothelial function. In contrast, the anti-inflammatory activity of HDL improves after consumption of polyunsaturated fat. These findings highlight novel mechanisms by which different dietary fatty acids may influence key atherogenic processes.

My take is that this is not the first study to find that satured fat is more atherogenic than several other FA types.
 
BTW I've already seen studies that use hydrogenated coconut oil as a SFA agent which obviously throws things off, I don't know about this study.
 
BTW I've already seen studies that use hydrogenated coconut oil as a SFA agent which obviously throws things off, I don't know about this study.

How is it possible to hydrogenate saturated fats? I thought that since its saturated, you can't add any more hydrogens?
 
I dont know but go compare the organic unprocessed coconut oil to the shit in wal mart and tell me which one is gud 4 u
 
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