C-19 is asymptomatic in too many people for the idea that more testing can eliminate it. Impossible.

[Amerikuracana]

The Dems hearts are possibly in the right place on this, and without deep thinking it sounds like an OK idea. But it's not living in reality, and if they ignore reality and use improper language for political purposes then their hearts are no longer in the right place. I've been starting to see some political propaganda saying "Do these things so we can eradicate this virus." It's misleading, and it's going to be misused.

The only way massive forced testing would work is if you got everybody (100%) tested, and then literally disintegrated all those who tested positive within 10 seconds of the positive test. That is literally the only way that 30 billion for testing could "eradicate" this disease.

Sometimes there are no perfect solutions, and more testing can be a good idea (I've advocated for it) for purposes of having an accurate picture about the deadliness of the virus itself, which I have predicted would end up being between .2 -.8% since the beginning based on the amount of people tested and the condition those being tested were in, with knowledge of the oft asymptomatic nature of the infection. So yes to more testing, but a big NO to the propaganda notion that more testing is needed to (or even potentially could) "defeat" the virus. That's a completely pointless endeavor on a macro-national scale. Of course finding out early when at-risk people have the disease could assist in their treatment. Of course more testing could possibly stop outbreaks on a micro-scale in nursing homes and other at-risk facilities. If either side starts talking about "Eradicating the virus" through any of these measures, or any amount of testing, they are lying to you for politics. Straight lying.

More general testing is a good idea, but you absolutely are not going to be able to test every person, and then fully quarantine every infected person from every non-infected person. Impossible, out of the question, and completely futile if attempted. It was (allegedly) TWO people that allegedly brought this to Italy. TWO. Although I personally have doubts about that exact number, the fact remains that this spreads like a cold. Two people being asymptomatic and not getting tested restart the entire thing, so if you pretend you can make it "disappear" by partially shutting down the economy, like I saw in a meme with nurses holding up signs today, you are spreading dangerous false notions. The testing needs to be for (and sold to us as) a way to try and protect at risk people, and to get early treatment for those with beginning stages of worsening non-asymptomatic symptoms. Not as a way to "eradicate" Covid, or make it "disappear."

This is reality.


P.S. Oh, has anybody noticed that a bunch of Doctors and scientists have now done appropriate balancing tests with available data, and realized that a great depression level destruction of the economy would definitely kill more people early than the virus itself? Gee.. Who said that first and had morons make moronic comments about it?

 

[Queen B]

It makes testing that much more important

 

[7437]

It works with contact tracing. That's the point of how places like south Korea did it.

Just testing everyone misses the point and is kinda a dumb idea. We need expanded testing of anyone who has been in contact. The number of asymptomatic people means that testing only sick people is pointless.

 

[Tycho- Taylor's Version]

You know, when you come up with theory of what someone else thinks that sounds really, really stupid, you have 2 options:

1) Said someone else is really stupid.

2) You have a really stupid perception of what they think.

Maybe test out number two in this case.

 

[Amerikuracana]

It works with contact tracing. That's the point of how places like south Korea did it.

Just testing everyone misses the point and is kinda a dumb idea. We need expanded testing of anyone who has been in contact. The number of asymptomatic people means that testing only sick people is pointless.

Contact tracing had some positive effects over there, but it would not do much at all here at this point. It's a different situation, different geography, and different population. It wouldn't work. I don't hate the notion or anything, but I'm being realistic. It's going to become a political buzz-phrase, I know.

The asymptomatic nature, and not doing this very, very early makes contact tracing nearly pointless if started now or in the near future. SK got to it really early and made a difference. We aren't there.

 

[Brass Balls]

Remember when it was shut everything down until the curve is flattened. Now it's sounding like the goal has changed to such extremes as much as shut everything down until the virus is gone....
Almost like some global "leaders" are playing politics & trying to destroy the economy.
This illness can be deadly to some of the population but things don't add up to the level of hysteria.

 

[SBJJ]

Way way way too late for contact tracing in heavy hit areas. Let's be real, if the studies coming out on infections is correct, a place like NYC is probably at 60% infected

 

[THEfightsAREfixed]

there is no experimental evidence demonstrating asymptomatic transmission of corona virus, whether it be SARS-CoV-1 or SARS-CoV-2, also please stop calling it covid-19 thats a made up name for the symptoms of any general respiratory viral infection but actually has no testable definition, you can only test for SARS-CoV-2 through a few different methods and all of them are susceptible to cross-reactivity from other corona virus antibodies, providing false positives for SARS-CoV-2 at like a rate of 1 in 7 (as demonstrated by SARS-CoV-1). There are four common corona viruses that cause respiratory illness ( HCoV-229E, HCoV-HKU1, HCoV-NL63, HCoV-OC43), and all corona viruses are associated with high coinfection rates, meaning when you test for SARS-CoV-2 antibodies you are also testing additional coronaviruses in the sample that will provide false positives

heres what i could gather and attempt to analyze this morning for a few hours, in case anyone is interested at how at arrived at the aforementioned conclusions and assertions, good fucking luck though this will involve actually using your fucking brain instead of just drinking up whatever ridiculous shit pops up on the TV


initial analysis of what becomes SARS-CoV-2

https://www.nature.com/articles/s41586-020-2008-3



inventing the term "covid-19" and using the terminology "disease"

https://www.who.int/dg/speeches/det...dia-briefing-on-2019-ncov-on-11-february-2020

(no medical research refers to covid-19 or as a "disease", only as SARS-CoV-2 virus and viral infection)





SARS-COVID-(1) = 2003 virus pandemic in China





SARS-COVID-2 **COVID 19** = 2020 virus global pandemic


MERS-COVID-1 = MIddle east virus pandemic 2012


MERS

https://en.wikipedia.org/wiki/Middle_East_respiratory_syndrome-related_coronavirus

(Initially called simply novel coronavirus or nCoV, it was first reported in 2012 after genome sequencing of a virus isolated from sputum samples from a person who fell ill in a 2012 outbreak of a new flu-like respiratory illness.)

1. 229E (alpha coronavirus)
   
   
2. NL63 (alpha coronavirus)
   
   
3. OC43 (beta coronavirus)
   
   
4. HKU1 (beta coronavirus)
   

*Corona viruses that cause "cold" or respiratory illness, 7 CoVs are known to cause respiratory illness ; coronaviruses = common cold



(there is no proven mechanism for asymptomatic transmission, only contagious with symptoms, zero documented and proven asymptomatic transmission cases for SARS-CoV-1 or 2 ) 2020

https://jamanetwork.com/journals/jama/article-abstract/2762028

"One previous study reported an asymptomatic 10-year-old boy with COVID-19 infection, but he had abnormalities on chest CT.6 If the findings in this report of presumed transmission by an asymptomatic carrier are replicated, the prevention of COVID-19 infection would prove challenging. The mechanism by which asymptomatic carriers could acquire and transmit the coronavirus that causes COVID-19 requires further study."







https://www.cdc.gov/coronavirus/types.html

"People around the world commonly get infected with human coronaviruses 229E, NL63, OC43, and HKU1."



https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2916580/#r26 2010

"All coronaviruses are associated with high coinfection rates..."



(exact scenario for SARS-COVID-1 or 2, but actually another corona virus) 2006

https://www.ncbi.nlm.nih.gov/pubmed/18382647

"These findings underscore the virulence of human CoV-OC43 in elderly populations and confirm that cross-reactivity to antibody against nucleocapsid proteins from these viruses must be considered when interpreting serological tests for SARS-CoV."



(there are a variety of corona viruses similar to SARS-COVID-2) 2010

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2916580/#r36

" Four human coronaviruses (human coronavirus 229E [HCoV-229E], HCoV-HKU1, HCoV-NL63, and HCoV-OC43) are associated with a range of respiratory symptoms, including high-morbidity outcomes such as pneumonia and bronchiolitis "





(antibody tests are not 100% accurate, maybe over 10% false positive for other antibody)

https://www.ncbi.nlm.nih.gov/pubmed/15583332 2004

https://www.ncbi.nlm.nih.gov/pubmed/15897988 2005

"Three of the 21 and 1 of the 7 convalescent-phase serum samples from persons with increases in antibodies against HCoV-OC43 and HCoV-229E, respectively, tested positive by the recombinant SARS-CoV nucleocapsid protein-based ELISA. None of these samples were found to contain a specific antibody in the recombinant SARS-CoV spike polypeptide-based Western blot assay."

"Cross-reactivity between antibodies to different human coronaviruses (HCoVs) has not been systematically studied. " <---- false positives for SARS-CoV-2





**(precedent for bat meme)** 2013

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3747529/

"However, the source of SARS virus remains a myth.” A recent report denied the role of civets in transmitting SARS virus and argued that bat is the real culprit. According to Dr. He, however, this is not a new story: early in 2005, some scientists had made the same conclusion. However, up to now there has been no final conclusion. "











developing advanced techniques for corona virus splicing as a result of SARS 2014

https://www.sciencedirect.com/science/article/pii/S0168170214002408?via=ihub

"The potential risk to public health posed by SARS-CoV and other CoVs, and the lack of specific antiviral agents and vaccines, have triggered a global effort to study this family of viruses at the molecular level in order to develop effective strategies to prevent and control CoV infections. Molecular genetic analysis of the structure and function of RNA virus genomes has been profoundly advanced by the availability of full-length cDNA clones. In the case of CoV, the huge genome size and the instability of specific CoV cDNA sequences in bacterial systems hindered the development of infectious cDNA clones until recently. A tremendous amount of creativity in the CoV field resulted in the development of four independent and unique reverse genetic systems that overcame these problems. These reverse genetic systems have been established using non-traditional approaches, which are based on the use of targeted recombination, BACs, in vitro ligation of CoV cDNA fragments, and vaccinia virus as a vector for the propagation of CoV genomic cDNAs. The availability of CoV full-length infectious clones and recombinant viruses expressing reporter genes constitute important tools for the study of CoV replication and transcription mechanisms, virus-host interaction and pathogenesis, and also for the rapid and rational development and testing of genetically defined vaccines. In addition to full-length cDNA clones, the generation of CoV replicons has greatly facilitated the functional analysis of viral replication and transcription, as well as the analysis of antiviral drugs without the need of manipulating infectious viruses."


collecting corona viruses from bats 2018

https://www.tandfonline.com/doi/full/10.1038/s41426-018-0155-5

 

[Seano]

Isn't that exactly why testing is needed? You're positive even if asymptomatic, aren't you?

 

[Amerikuracana]

Way way way too late for contact tracing in heavy hit areas. Let's be real, if the studies coming out on infections is correct, a place like NYC is probably at 60% infected

And it would actually be easier in those densely populated areas, even though still next to impossible. When you start talking about how expansive the United States is it becomes clear that A. You are correct that it is too late in areas dense enough to at one point maybe be possible, and B. The differences in the spread of the disease and America's geography would render it completely useless even if tried a month ago.

 

[Amerikuracana]

Isn't that exactly why testing is needed? You're positive even if asymptomatic, aren't you?

More testing isn't a bad idea to keep as many as possible out of the nursing homes if positive, but that's about it.

 

[chardog]

Isn't that exactly why testing is needed? You're positive even if asymptomatic, aren't you?

Early testing, it would give inaccurate results. Not clear if this has changed since.

the amount of time it takes was another issue. At this point, I can’t really think of anything other than herd immunity. Of course, the longer we delay the game, better to find more definitive treatment at different stages. There’s also the issue of people losing it and going on protests. And then violence.

 

[Amerikuracana]

there is no experimental evidence demonstrating asymptomatic transmission of corona virus, whether it be SARS-CoV-1 or SARS-CoV-2, also please stop calling it covid-19 thats a made up name for the symptoms of any general respiratory viral infection but actually has no testable definition, you can only test for SARS-CoV-2 through a few different methods and all of them are susceptible to cross-reactivity from other corona virus antibodies, providing false positives for SARS-CoV-2 at like a rate of 1 in 7 (as demonstrated by SARS-CoV-1). There are four common corona viruses that cause respiratory illness ( HCoV-229E, HCoV-HKU1, HCoV-NL63, HCoV-OC43), and all corona viruses are associated with high coinfection rates, meaning when you test for SARS-CoV-2 antibodies you are also testing additional coronaviruses in the sample that will provide false positives

heres what i could gather and attempt to analyze this morning for a few hours, in case anyone is interested at how at arrived at the aforementioned conclusions and assertions, good fucking luck though this will involve actually using your fucking brain instead of just drinking up whatever ridiculous shit pops up on the TV


initial analysis of what becomes SARS-CoV-2

https://www.nature.com/articles/s41586-020-2008-3



inventing the term "covid-19" and using the terminology "disease"

https://www.who.int/dg/speeches/det...dia-briefing-on-2019-ncov-on-11-february-2020

(no medical research refers to covid-19 or as a "disease", only as SARS-CoV-2 virus and viral infection)





SARS-COVID-(1) = 2003 virus pandemic in China





SARS-COVID-2 **COVID 19** = 2020 virus global pandemic


MERS-COVID-1 = MIddle east virus pandemic 2012


MERS

https://en.wikipedia.org/wiki/Middle_East_respiratory_syndrome-related_coronavirus

(Initially called simply novel coronavirus or nCoV, it was first reported in 2012 after genome sequencing of a virus isolated from sputum samples from a person who fell ill in a 2012 outbreak of a new flu-like respiratory illness.)

1. 229E (alpha coronavirus)
   
   
2. NL63 (alpha coronavirus)
   
   
3. OC43 (beta coronavirus)
   
   
4. HKU1 (beta coronavirus)

*Corona viruses that cause "cold" or respiratory illness, 7 CoVs are known to cause respiratory illness ; coronaviruses = common cold



(there is no proven mechanism for asymptomatic transmission, only contagious with symptoms, zero documented and proven asymptomatic transmission cases for SARS-CoV-1 or 2 ) 2020

https://jamanetwork.com/journals/jama/article-abstract/2762028

"One previous study reported an asymptomatic 10-year-old boy with COVID-19 infection, but he had abnormalities on chest CT.6 If the findings in this report of presumed transmission by an asymptomatic carrier are replicated, the prevention of COVID-19 infection would prove challenging. The mechanism by which asymptomatic carriers could acquire and transmit the coronavirus that causes COVID-19 requires further study."







https://www.cdc.gov/coronavirus/types.html

"People around the world commonly get infected with human coronaviruses 229E, NL63, OC43, and HKU1."



https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2916580/#r26 2010

"All coronaviruses are associated with high coinfection rates..."



(exact scenario for SARS-COVID-1 or 2, but actually another corona virus) 2006

https://www.ncbi.nlm.nih.gov/pubmed/18382647

"These findings underscore the virulence of human CoV-OC43 in elderly populations and confirm that cross-reactivity to antibody against nucleocapsid proteins from these viruses must be considered when interpreting serological tests for SARS-CoV."



(there are a variety of corona viruses similar to SARS-COVID-2) 2010

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2916580/#r36

" Four human coronaviruses (human coronavirus 229E [HCoV-229E], HCoV-HKU1, HCoV-NL63, and HCoV-OC43) are associated with a range of respiratory symptoms, including high-morbidity outcomes such as pneumonia and bronchiolitis "





(antibody tests are not 100% accurate, maybe over 10% false positive for other antibody)

https://www.ncbi.nlm.nih.gov/pubmed/15583332 2004

https://www.ncbi.nlm.nih.gov/pubmed/15897988 2005

"Three of the 21 and 1 of the 7 convalescent-phase serum samples from persons with increases in antibodies against HCoV-OC43 and HCoV-229E, respectively, tested positive by the recombinant SARS-CoV nucleocapsid protein-based ELISA. None of these samples were found to contain a specific antibody in the recombinant SARS-CoV spike polypeptide-based Western blot assay."

"Cross-reactivity between antibodies to different human coronaviruses (HCoVs) has not been systematically studied. " <---- false positives for SARS-CoV-2





**(precedent for bat meme)** 2013

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3747529/

"However, the source of SARS virus remains a myth.” A recent report denied the role of civets in transmitting SARS virus and argued that bat is the real culprit. According to Dr. He, however, this is not a new story: early in 2005, some scientists had made the same conclusion. However, up to now there has been no final conclusion. "











developing advanced techniques for corona virus splicing as a result of SARS 2014

https://www.sciencedirect.com/science/article/pii/S0168170214002408?via=ihub

"The potential risk to public health posed by SARS-CoV and other CoVs, and the lack of specific antiviral agents and vaccines, have triggered a global effort to study this family of viruses at the molecular level in order to develop effective strategies to prevent and control CoV infections. Molecular genetic analysis of the structure and function of RNA virus genomes has been profoundly advanced by the availability of full-length cDNA clones. In the case of CoV, the huge genome size and the instability of specific CoV cDNA sequences in bacterial systems hindered the development of infectious cDNA clones until recently. A tremendous amount of creativity in the CoV field resulted in the development of four independent and unique reverse genetic systems that overcame these problems. These reverse genetic systems have been established using non-traditional approaches, which are based on the use of targeted recombination, BACs, in vitro ligation of CoV cDNA fragments, and vaccinia virus as a vector for the propagation of CoV genomic cDNAs. The availability of CoV full-length infectious clones and recombinant viruses expressing reporter genes constitute important tools for the study of CoV replication and transcription mechanisms, virus-host interaction and pathogenesis, and also for the rapid and rational development and testing of genetically defined vaccines. In addition to full-length cDNA clones, the generation of CoV replicons has greatly facilitated the functional analysis of viral replication and transcription, as well as the analysis of antiviral drugs without the need of manipulating infectious viruses."


collecting corona viruses from bats 2018

https://www.tandfonline.com/doi/full/10.1038/s41426-018-0155-5

Well some of that is relevant, but not all. I disagree that the spread is only from people showing symptoms. That's ridiculous to me. It just gets spread more easily from those people because they are coughing and leaking fluids. The speed at which it spreads makes me fairly certain that people with the virus, can spread the virus. If you did a study with a small sample size you could get the false results that asymptomatics did not transmit because they do so significantly less.

 

[SBJJ]

And it would actually be easier in those densely populated areas, even though still next to impossible. When you start talking about how expansive the United States is it becomes clear that A. You are correct that it is too late in areas dense enough to at one point maybe be possible, and B. The differences in the spread of the disease and America's geography would render it completely useless even if tried a month ago.

You'd also need a team of at least half a million for the job.

 

[Amerikuracana]

You'd also need a team of at least half a million for the job.

Yes. It would be a ridiculous and unsustainable growth in the government bureaucracy at a time when sentiment from The People is to shrink it.

 

[KONG-D'SNT-TAP]

Oh good this dope who isn’t a scientist or doctor has it all figured out. I can rest easy tonight.

 

[Amerikuracana]

Oh good this dope who isn’t a scientist or doctor has it all figured out. I can rest easy tonight.

Don't worry.. The Medical Doctors will pick up on it in about a week, and you'll hear it then. Sleep well.

I do have a Doctorate, btw.

 

[kenetics]

there is no experimental evidence demonstrating asymptomatic transmission of corona virus, whether it be SARS-CoV-1 or SARS-CoV-2, also please stop calling it covid-19 thats a made up name for the symptoms of any general respiratory viral infection but actually has no testable definition, you can only test for SARS-CoV-2 through a few different methods and all of them are susceptible to cross-reactivity from other corona virus antibodies, providing false positives for SARS-CoV-2 at like a rate of 1 in 7 (as demonstrated by SARS-CoV-1). There are four common corona viruses that cause respiratory illness ( HCoV-229E, HCoV-HKU1, HCoV-NL63, HCoV-OC43), and all corona viruses are associated with high coinfection rates, meaning when you test for SARS-CoV-2 antibodies you are also testing additional coronaviruses in the sample that will provide false positives

heres what i could gather and attempt to analyze this morning for a few hours, in case anyone is interested at how at arrived at the aforementioned conclusions and assertions, good fucking luck though this will involve actually using your fucking brain instead of just drinking up whatever ridiculous shit pops up on the TV


initial analysis of what becomes SARS-CoV-2

https://www.nature.com/articles/s41586-020-2008-3



inventing the term "covid-19" and using the terminology "disease"

https://www.who.int/dg/speeches/det...dia-briefing-on-2019-ncov-on-11-february-2020

(no medical research refers to covid-19 or as a "disease", only as SARS-CoV-2 virus and viral infection)





SARS-COVID-(1) = 2003 virus pandemic in China





SARS-COVID-2 **COVID 19** = 2020 virus global pandemic


MERS-COVID-1 = MIddle east virus pandemic 2012


MERS

https://en.wikipedia.org/wiki/Middle_East_respiratory_syndrome-related_coronavirus

(Initially called simply novel coronavirus or nCoV, it was first reported in 2012 after genome sequencing of a virus isolated from sputum samples from a person who fell ill in a 2012 outbreak of a new flu-like respiratory illness.)

1. 229E (alpha coronavirus)
   
   
2. NL63 (alpha coronavirus)
   
   
3. OC43 (beta coronavirus)
   
   
4. HKU1 (beta coronavirus)

*Corona viruses that cause "cold" or respiratory illness, 7 CoVs are known to cause respiratory illness ; coronaviruses = common cold



(there is no proven mechanism for asymptomatic transmission, only contagious with symptoms, zero documented and proven asymptomatic transmission cases for SARS-CoV-1 or 2 ) 2020

https://jamanetwork.com/journals/jama/article-abstract/2762028

"One previous study reported an asymptomatic 10-year-old boy with COVID-19 infection, but he had abnormalities on chest CT.6 If the findings in this report of presumed transmission by an asymptomatic carrier are replicated, the prevention of COVID-19 infection would prove challenging. The mechanism by which asymptomatic carriers could acquire and transmit the coronavirus that causes COVID-19 requires further study."







https://www.cdc.gov/coronavirus/types.html

"People around the world commonly get infected with human coronaviruses 229E, NL63, OC43, and HKU1."



https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2916580/#r26 2010

"All coronaviruses are associated with high coinfection rates..."



(exact scenario for SARS-COVID-1 or 2, but actually another corona virus) 2006

https://www.ncbi.nlm.nih.gov/pubmed/18382647

"These findings underscore the virulence of human CoV-OC43 in elderly populations and confirm that cross-reactivity to antibody against nucleocapsid proteins from these viruses must be considered when interpreting serological tests for SARS-CoV."



(there are a variety of corona viruses similar to SARS-COVID-2) 2010

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2916580/#r36

" Four human coronaviruses (human coronavirus 229E [HCoV-229E], HCoV-HKU1, HCoV-NL63, and HCoV-OC43) are associated with a range of respiratory symptoms, including high-morbidity outcomes such as pneumonia and bronchiolitis "





(antibody tests are not 100% accurate, maybe over 10% false positive for other antibody)

https://www.ncbi.nlm.nih.gov/pubmed/15583332 2004

https://www.ncbi.nlm.nih.gov/pubmed/15897988 2005

"Three of the 21 and 1 of the 7 convalescent-phase serum samples from persons with increases in antibodies against HCoV-OC43 and HCoV-229E, respectively, tested positive by the recombinant SARS-CoV nucleocapsid protein-based ELISA. None of these samples were found to contain a specific antibody in the recombinant SARS-CoV spike polypeptide-based Western blot assay."

"Cross-reactivity between antibodies to different human coronaviruses (HCoVs) has not been systematically studied. " <---- false positives for SARS-CoV-2





**(precedent for bat meme)** 2013

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3747529/

"However, the source of SARS virus remains a myth.” A recent report denied the role of civets in transmitting SARS virus and argued that bat is the real culprit. According to Dr. He, however, this is not a new story: early in 2005, some scientists had made the same conclusion. However, up to now there has been no final conclusion. "











developing advanced techniques for corona virus splicing as a result of SARS 2014

https://www.sciencedirect.com/science/article/pii/S0168170214002408?via=ihub

"The potential risk to public health posed by SARS-CoV and other CoVs, and the lack of specific antiviral agents and vaccines, have triggered a global effort to study this family of viruses at the molecular level in order to develop effective strategies to prevent and control CoV infections. Molecular genetic analysis of the structure and function of RNA virus genomes has been profoundly advanced by the availability of full-length cDNA clones. In the case of CoV, the huge genome size and the instability of specific CoV cDNA sequences in bacterial systems hindered the development of infectious cDNA clones until recently. A tremendous amount of creativity in the CoV field resulted in the development of four independent and unique reverse genetic systems that overcame these problems. These reverse genetic systems have been established using non-traditional approaches, which are based on the use of targeted recombination, BACs, in vitro ligation of CoV cDNA fragments, and vaccinia virus as a vector for the propagation of CoV genomic cDNAs. The availability of CoV full-length infectious clones and recombinant viruses expressing reporter genes constitute important tools for the study of CoV replication and transcription mechanisms, virus-host interaction and pathogenesis, and also for the rapid and rational development and testing of genetically defined vaccines. In addition to full-length cDNA clones, the generation of CoV replicons has greatly facilitated the functional analysis of viral replication and transcription, as well as the analysis of antiviral drugs without the need of manipulating infectious viruses."


collecting corona viruses from bats 2018

https://www.tandfonline.com/doi/full/10.1038/s41426-018-0155-5

Too long, did not read .

 

[KONG-D'SNT-TAP]

Don't worry.. The Medical Doctors will pick up on it in about a week, and you'll hear it then. Sleep well.

I do have a Doctorate, btw.

Brah you’ve been batting 0 since this started. You’ve struck out every single time you’ve been up to the plate.


Stick with our shitty short stories in the Mayberry. They’re somehow less shitty than this nonsense.

 

[THEfightsAREfixed]

Well some of that is relevant, but not all. I disagree that the spread is only from people showing symptoms. That's ridiculous to me. It just gets spread more easily from those people because they are coughing and leaking fluids. The speed at which it spreads makes me fairly certain that people with the virus, can spread the virus. If you did a study with a small sample size you could get the false results that asymptomatics did not transmit because they do so significantly less.

There is zero empirical or experimental data proving that asymptomatic transmission has occured, and the mechanism for which it could is unknown, if you want to believe random shit because it fits a story in your head thats your prerogative